Abstract
Introduction: Physical exercise may be an important adjunct to pharmacological and other non-pharmacological treatment of Alzheimer s disease (AD). Several studies have indicated a beneficial effect of physical activity on symptoms of AD, but biological mechanisms underlying this effect remains relatively uninvestigated. Such mechanisms include induction of neurogenesis, effects on vascular risk factors as well as reduction of deposits of beta-amyloid. The objectives of the present study was to investigate whether moderate-high intensity aerobic exercise reduces the accumulation of beta-amyloid in AD.
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