Abstract
IT is now known that a number of insect species which are resistant to insecticides possess effective means of detoxication in order to protect themselves from being exposed to the toxicant at the vital site1–3. One of these insects, Culex tarsalis Fresno strain, was shown to have developed a malathion Carboxyesterase3 with a very narrow substrate specificity against malathion analogues4. This agrees perfectly with the fact that this strain shows little cross-resistance to any other organophosphorus insecticides.
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