Abstract
Stimulus-frequency otoacoustic emissions (SFOAEs) have been used in previous research to estimate cochlear tuning in humans. These estimates of tuning rely on the theory that SFOAEs arise from coherent reflections from the place on the basilar membrane (BM) with the characteristic frequency (CF) of the tone evoking the emission. Theories underlying SFOAE generation are still the subject of much debate, and several recent studies have shown evidence supporting an alternative theory postulating that generators of SFOAE are distributed basally to the CF place on the BM. Basally distributed emission generators could explain why SFOAE-derived group delays measured in the chinchilla cochlea for low-frequency tones were shorter than predicted by coherent-reflection hypothesis. The aim of the current study is to look for potential effects of basal emission generators in SFOAE-derived measurements of group delay at low frequencies in humans. SFOAE group delays were measured at 0.5, 0.75 kHz with and without a low intensity highpass noise, used to perturb basally distributed emission generation sites. Results show that the presence of the highpass noise leads to increased group delays estimated from SFOAEs, consistent with the hypothesis that basal generators of emission influence estimates of cochlear group delay at low frequencies.
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