Abstract
The mechanism of antiischemic effect of low-energy laser radiation was studied in rats with experimental myocardial infarction taking into consideration the effect of laser on hemoglobin affinity for oxygen and intensity of lipid peroxidation. Low-energy laser irradiation for 15 min in vitro decreased the area of myocardial infarction, slightly reduced hemoglobin affinity for oxygen, and inhibited lipid peroxidation. Short-term low-energy laser irradiation did not reduce the area of necrosis, initiated lipid peroxidation, and increased superoxide dismutase activity.
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