Abstract

BackgroundImplantoplasty reduces both implant diameter and the thickness of its walls, subsequently reducing the ability of the implant to resist fracture in response to functional load. In combination with an increase in the crown-implant ratio due to bone loss, this could increase the lever effect, which in presence of high masticatory forces or parafunctional habits, could lead to complications such as fracture of the implant or loosening of the prosthetic screw.ObjectivesTo determine the elastic limits of internal connection, dental implants of different designs and diameters after an implantoplasty.Materials and methodsThis in vitro study included 315 tapered internal connection titanium dental implants, the threads of which were removed with an industrial milling machine—for standardized implantoplasty (IMP1; n = 105)—or with the conventional approach—manually, using high-speed burs (IMP2; n = 105). The remaining 105 implants were used as controls. The final implant diameters were recorded. The quality of the newly polished surfaces was assessed by scanning electron microscopy. All implants were subjected to a mechanical pressure resistance test. A Tukey’s test for multiple comparisons was used to detect differences in the elastic limit and final implant diameters between the implant groups.ResultsThere were statistically significant differences in the elastic limit between the IMP1, IMP2, and control groups (p < 0.05). Furthermore, the implant diameter was significantly smaller in the IMP1 and IMP2 groups (p < 0.05). Scanning electron microscopy revealed smooth implant surfaces in the IMP1 and IMP2 groups, with some titanium particles visible in the IMP1 group.ConclusionsImplantoplasty significantly decreased the elastic limit of internal connection titanium dental implants, especially in those with a smaller diameter (3-3.5 mm).

Highlights

  • Implantoplasty reduces both implant diameter and the thickness of its walls, subsequently reducing the ability of the implant to resist fracture in response to functional load

  • Implantoplasty significantly decreased the elastic limit of internal connection titanium dental implants, especially in those with a smaller diameter (3-3.5 mm)

  • Mucositis affects only the periimplant mucosa and does not involve the marginal bone, whereas peri-implantitis is a pathological plaqueassociated condition characterized by inflammation of the peri-implant mucosa and subsequent progressive loss of supporting bone [3]

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Summary

Introduction

Implantoplasty reduces both implant diameter and the thickness of its walls, subsequently reducing the ability of the implant to resist fracture in response to functional load. The dental implants replacing missing teeth have been reported to have a long-term survival rate of 96.4% [1]. They may be associated with esthetic, mechanical, or biological complications, including mucositis and peri-implantitis [2]. Mucositis affects only the periimplant mucosa and does not involve the marginal bone, whereas peri-implantitis is a pathological plaqueassociated condition characterized by inflammation of the peri-implant mucosa and subsequent progressive loss of supporting bone [3]. Peri-implantitis is an important cause of implant failure, affecting up to 18.5% of treated patients and 12.8% of all dental implants placed, according to a recent meta-analysis [4]. The mechanical, non-surgical treatment seems to be ineffective [8,9,10,11], and by far, surgical treatment is considered the best option for the treatment of moderate or severe cases [12]

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