Abstract
The experimental chemical, N-[2-(2-oxo-1-imidazolidinyl)ethyl]- N-phenylurea (Ethylene diurea, EDU) has been reported to protect plants from otherwise injurious doses of the air pollutant, ozone. It has been claimed that EDU provides this protection by causing enhancement of the levels of the oxyradical scavenger, superoxide dismutase (SOD). Studies with Phaseolus vulgaris L. cv Pure Goldwax have confirmed the protective effect of EDU against acute ozone doses which otherwise resulted in visible injury to more than 70% of the area of leaves on untreated plants. However, no significant effects of EDU treatment were observed on SOD levels, in either primary or trifoliate leaves. In this and other cultivars, EDU appears to function via a mechanism which does not involve SOD. Evidence is presented to show that the lack of treatment effects was not the result of possible SOD inactivation during extraction.
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