Abstract
In the modern view nociceptive sensation is the integrated response of the CNS invariably involving cortico-subcortical mechanisms of interaction [2, 7, 8]. Animal experiments have shown that the second somatosensory cortical area (SII) plays the leading role in evaluation of the extremal significance of incoming information (including nociceptive) at the cortical level [3]. It has been suggested that a reduction in the intensity of pain sensations associated with EAP may be largely due to a change in the character of cortico-subcortical interactions in the direction of strengthening of the mechanism of cortical inhibition [5].
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