Abstract
We have previously reported that experimental cerebral concussion in primates subjected to sagittal plane angular acceleration of the head was associated with a rapid, marked increase in intracranial pressure (ICP) and a parallel rise in mean arterial blood pressure (MAP) (Gennarelli et al. 1980). The present study of coronal plane angular acceleration shows that, for an equivalent severity of injury, these changes to do not occur. The findings indicate that cerebral concussion may be produced without a dramatic rise in ICP and that the direction of head movement during acceleration injury is an important determinant of this physiological response.
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