Abstract
An apparent paradox in thyroidal economy is presented by the normally pregnant woman. Thus, there occurs during pregnancy thyroidal hyperplasia, augmentation of the thyroidal accumulation of radioiodine, and an increase in the concentration of circulating thyroid hormone (1–5). These changes, ordinarily associated with the thyrotoxic state, are not accompanied during pregancy by symptomatic stigmata of thyrotoxicity nor by increases in basal oxygen consumption beyond those which have been attributed to fetal needs (6).
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