Abstract
The role of Ca 2+ in glycerol dissimilation under hypoosmotic stress in the halotolerant alga Dunaliella tertiolecta was investigated using a pharmacological approach. A stretch-activated Ca 2+ channel blocker, GdCl 3, inhibited glycerol dissimilation under hypoosmotic stress. However, addition of voltage-dependent Ca 2+ channel blockers and inhibitors of mitochondrial and endoplasmic reticulum Ca 2+ channels did not affect the glycerol dissimilation under hypoosmotic stress. The results of the present study suggest that the influx of Ca 2+ from the extracellular space via the stretch-activated Ca 2+ channels localized in the plasma membrane is required for the transduction of osmotic signal of D. tertiolecta.
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