Abstract
Aromatase P450 is the key enzyme for estrogen biosynthesis (Bulun SE et al. Semin Reprod Med.;22:45–50, 2004) and has been found to be overly expressed in eutopic endometrium from patients with endometriosis (EDT). The aromatase inhibitor Letrozole (Let) has recently been proposed for the treatment of EDT in reproductive age women (Ailawadi RK, Fertil Steril.;81:290–6, 2004). Our purpose was to study the effect of Let on apoptosis and cell proliferation in epithelial endometrial cells (EEC) from patients with EDT.
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