Abstract
Physical exercise elicits an increase in heart rate (HR), blood pressure (BP) and, consequently, in the rate-pressure product (RPP). Recovery of HR immediately after exercise indicates cardiovascular health. Blood pressure also decreases after exercise, occasionally reaching values lower than pre-exercise levels (postexercise hypotension). Studies have shown a positive effect of water intake on HR recovery after exercise. However, little is known about the effect of water intake on postexercise BP and RPP responses. The objective of this study was to evaluate the effects of water intake on postexercise cardiac work assessed by HR, BP and RPP. Fourteen healthy volunteers (22 ± 1.4 years) participated in the study. The experimental session consisted of HR, systolic (SBP) and diastolic BP (DBP) recording at rest, followed by submaximal exercise on a cycle ergometer. Next, the subjects consumed water and the cardiovascular variables were recorded during recovery. In addition, a control session without postexercise water intake was performed. The RPP was calculated from the product of HR and SBP. Water intake prevented a postexercise hypotensive effect on DBP, but accelerated postexercise HR and RPP reduction during recovery when compared to the control session. It was concluded that water intake is an effective strategy to reduce postexercise cardiac work.
Highlights
Intense physical activity elicits an increase in heart rate (HR) and blood pressure (BP) and, in myocardial oxygen consumption
When compared to resting BP values, no differences in postexercise systolic BP (SBP) or diastolic BP (DBP) were observed at any time of recovery after the water intake session
No differences in the rate-pressure product (RPP) were observed between the water intake and control sessions throughout the recovery period (Figure 1D)
Summary
Intense physical activity elicits an increase in heart rate (HR) and blood pressure (BP) and, in myocardial oxygen consumption. Studies have shown that water intake promotes an increase in BP through sympathetic activation[7], a response that may inhibit PEH. In this respect, Endo et al.[8] investigated the effects of water intake on postexercise BP responses and observed the absence of PEH. The effect of water intake on the postexercise rate-pressure product (RPP) remains unknown. This variable – systolic BP (SBP) x HR – estimates myocardial work[10] and has an important prognostic value, with higher RPP values being associated with an increased risk of cardiovascular events[11]. The objective of the present study was to evaluate the effect of water intake on HR, BP and RPP responses during recovery after a session of intense exercise
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