Abstract
Delivery, among other things, entails a sudden cutoff of a steady flow of nutrients. The newborn has now to fend for itself until an adequate food supply is once again established. The sequence of events envisaged at present is as follows: a decrease in blood glucose concentration; an increase in the rate of glucagon release; an activation of liver and fat adenylcyclase; activation of glycogenolysis and lipolysis and, perhaps, gluconeogenesis followed by induction of key gluconeogenic enzymes, phosphoenolpyruvatecarboxykinase (PEPK), glucose-6-phosphatase and some transaminases, particularly tyrosine transaminase (TAT). In the rat, it seems that gluconeogenesis increases before glycogen breakdown.1 No data . . .
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