Abstract
Editorial: Cellular and Phenotypic Plasticity in Cancer.
Highlights
Specialty section: This article was submitted to Molecular and Cellular Oncology, a section of the journal Frontiers in Oncology
In normal and cancer cells alike, epithelial–mesenchymal transition (EMT) is regulated through signaling pathways [1], the outcome of which is dictated by the balance and cross-talk between the pathways as reviewed within this Research Topic [2]
EMT and EMTlike processes contribute greatly to tumor heterogeneity, the challenges of which are highlighted for colorectal cancer within this Research Topic [5]
Summary
Specialty section: This article was submitted to Molecular and Cellular Oncology, a section of the journal Frontiers in Oncology. Cellular and phenotypic plasticity is a key feature of development and normal function of cells within most multicellular organisms. This plasticity observed in most cell types is retained in cancer and can lead to opportunistic adaptation allowing therapeutic escape and acquisition of motile and invasive abilities that pose ongoing challenges for effective therapy.
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