Abstract

Japanese-B-encephalitis (JE) is a disease caused by theJapanese encephalitisvirus from the Flavivirus group, the Flaviviridae family. In Indonesia, JE cases have not been widely reported in both animals and humans, although encephalitis in humans is common. Yamanaka reported positive for anti-JE antibodies in 96 (6%) pigs in Tulungagung District. Environmental elements need to be identified to support the establishment of a healthy ecosystem to minimize the potential of JE transmission. This study aims to identify the environment that can support JE transmission and conceptualize possible approaches. The method is carried out by conducting environmental observations of risky environmental conditions in April November 2016 and searching for supporting literature. The results showed that there were many puddles in the environment of chicken and pig farms. Besides that, there were many open puddles of household waste in the residential areas. This place is the habitat forCulexSp. mosquitoes. Another research reported 19.05% of pigs who were positive for Ab JE in Tulungagung District. Environmental improvement can be done by cultivating fish that is resistant to dirty water, for example, catfish in ponds in chicken and pig farms and the construction of closed disposal facilities.

Highlights

  • Japanese-B-encephalitis (JE) is a disease caused by Japanese encephalitis virus, Flavivirus group, the Flaviviridae family.[1]

  • Tulungagung District has an area of 1,055.65 Km2 located at 111o43'-112o07 'East Longitude and 7o51'-8o18' South Latitude

  • Data confirmation with the Tulungagung District Animal Husbandry Office and Animal Husbandry officers in the field shows that there are far fewer pig breeders, what remains are farms that have quite a lot of pigs that are still surviving around the Brantas river

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Summary

Introduction

Japanese-B-encephalitis (JE) is a disease caused by Japanese encephalitis virus, Flavivirus group, the Flaviviridae family.[1] In humans, JE infection can cause inflammation of the brain that can be fatal. Humans and horses are the final target of the transmission cycle (dead-end) because viremia is so short that it is difficult to transmit to other people.[4]

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