Abstract
Environmental exposures during sensitive windows of development can reprogram normal physiological responses and alter disease susceptibility later in life. Genistein is a phytoestrogen and belongs to the category of isoflavones. Our previous studies demonstrated that neonatal exposure to the genistein increases uterine fibroid tumor incidence and multiplicity in Eker rats (Greathouse et al., 2012). However, the detailed mechanism regarding xenoestrogen-induced promotion of fibroid development is largely unknown.
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