Abstract
Searchable abstracts of presentations at key conferences in endocrinology ISSN 1470-3947 (print) | ISSN 1479-6848 (online)
Highlights
Studies suggest that disturbed thyroid hormones (TH) homeostasis may lead to increased cancer risk
Our results suggest there could be a potential role for monomeric thyroid-stimulating hormone (TSH) determination prior to treatment initiation for subclinical hypothyroidism, mainly in young patients
Differentiated thyroid cancer (DTC)
Summary
Studies suggest that disturbed thyroid hormones (TH) homeostasis may lead to increased cancer risk. In mammary and thyroid glands of MMI treated rats, strong induction of analyzed genes expression was observed, including ARNT, THRA, THRB, SLC16A2, SLC2A1, CTNNB1, NCOA1, HDAC1, CMYC, HIF1A upregulated in both tissues, and VIM, CDH1 and TPOupregulated only in thyroid glands. Fatigue occurred more frequently in patients ≥55 years old at treatment onset comparing to younger ones
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