Abstract

We recently pointed out that there is no evidence to support the commonly held view that there is something wrong with the brains of children who have great difficulty learning to read. In response, it was argued that dyslexia should be considered to be a neurodevelopmental disorder because of its potential to adversely affect quality of life, and because there are differences between the brains of people with different levels of reading skill. We agree with these two points, but they are irrelevant to the issue in question, because neither establishes the critical notion of disrupted neurodevelopment; that is, a brain fault. Differences between groups do not imply that any individuals are abnormal, and calling a brain improperly developed on the basis of cultural issues has absurd implications. Even calling brains atypical is unfounded because reference to typicality hinges on knowledge of the relevant distributions, which is currently lacking. Moreover, there is at present no obvious role for neurology- or neuroscience-based input for the critical issue of the assessment and remediation of the reading difficulties themselves. We reiterate our conclusion that there is, at the moment, no credible foundation to support the claim that dyslexia is a neurodevelopmental disorder.

Highlights

  • In our recent article [1] ( PR18), we pointed out that there is no evidence to support the commonly held view of dyslexia as a neurodevelopmental disorder

  • Fraga González, Karipidis, and Tijms [2] ( FKT18) have taken issue with our view, coming forth with a response purporting to establish the notion of dyslexia as a neurodevelopmental disorder

  • Before focusing on our points of disagreement, let us point out some very substantial points on which we believe we are in agreement with FKT18

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Summary

Introduction

In our recent article [1] ( PR18), we pointed out that there is no evidence to support the commonly held view of dyslexia as a neurodevelopmental disorder. There is no evidence to suggest that there is anything wrong with the brains of children who have great difficulty learning to read despite normal sensory function and educational opportunity. Fraga González, Karipidis, and Tijms [2] ( FKT18) have taken issue with our view, coming forth with a response purporting to establish the notion of dyslexia as a neurodevelopmental disorder. FKT18 make no novel points that would require us to revise or augment our arguments, we believe it is important to respond in order to clarify matters, as it seems that our original contribution has been insufficiently effective in dispelling certain fundamental misconceptions. In the present brief rejoinder, we can more successfully explain what the main issues and problems are

Points of Convergence
Further Implications and Objections
Conclusions
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