Abstract

We studied dynamics of blood lactate concentration during recovery period of short duration‐high intensity exercise in acute hypoxia or normoxia. University track and field athletes were recruited for single‐blind randomized crossover designed study. All subjects completed wingate test in both acute hypoxia (FiO2=0.145) and normoxia (FiO2=0.21). Two wingate tests were separated at least 48 hours. After 10 min cycling warm‐up (100 W), subjects took 5 min rests and then conducted 30 sec wingate tests. After the wingate test, subjects of both conditions took 30 min rests in normoxia. Blood samplings were conducted 1 min before and, 0.5, 1, 2, 3, 4, 5, 7, 10, 20 and 30 min after the wingate tests to examine the blood lactate concentration. Peak power output (PPO), mean power output (MPO) and fatigue index (FI) were not different in both condition (PPO; 984.3±49.3 W and 971.0±32.1 W, MPO; 674.3±37.4 W and 680.0±36.3 W, FI; 49.2±7.1% and 49.6±5.2%, normoxia and hypoxia respectively). The AUC of blood lactate, when examined pre to 10 min values, was significantly higher in acute hypoxia than normoxia (p<0.05). The rates of reduction of blood lactate concentration was higher in hypoxia than normoxia(p<0.05, 0.42±0.02 and 0.28±0.03 mmol/L/min, respectively). These results suggested that muscle glycogen breakdown was higher in acute hypoxia than normoxia in spite of same wingate test performances. It also suggests that lactate oxidation may be higher in acute hypoxic group when resting at normoxia.This abstract is from the Experimental Biology 2018 Meeting. There is no full text article associated with this abstract published in The FASEB Journal.

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