Abstract
Purpose: Absences are characterized by an abrupt onset and end of generalized 3–4Hz spike and wave discharges (GSW), accompanied by unresponsiveness. Although previous EEG-fMRI studies showed that thalamus, default mode areas and caudate nuclei are involved in absence seizures, the contribution of these regions throughout the ictal evolution of absences remains unclear. Furthermore animal models provide evidence that absences are initiated by a cortical focus with a secondary involvement of the thalamus. The aim of this study was to investigate dynamic changes during absences.
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