Abstract
1. 1. d-Xylose enters rings of chicken small intestine, in vitro, significantly faster and to a greater extent than its epimer at carbon-2, d-lyxose. Final distribution of lyxose is indistinguishable from the extracellular space. 2. 2. Xylose, but not lyxose entry exhibits saturation kinetics. 3. 3. Xylose, but not lyxose entry is blocked by phlorizin, a specific inhibitor of sugar active transport. 4. 4. Na + is required for xylose, but not for lyxose entry. The Na + gradient, medium/tissue, determines the direction of net xylose movement into or out of the tissues. 5. 5. Xylose accumulation against the gradient has not been demonstrated, but a portion of the entry mechanism for xylose is sensitive to dinitrophenol inhibition. 6. 6. In accord with the mobile-carrier concept, xylose countertransport (substrate-induced counterflow) can be demonstrated using actively transported sugars and analogs as elicitors. 7. 7. It is concluded that xylose transport in the chicken small intestine occurs through a Na +-dependent mobile carrier mechanism similar to that operating in mammals.
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