Abstract
To the Editor: We read with great interest the correspondence from Vitiello and Cammelli1, who described their experience on stroke occurring in patients newly diagnosed with giant cell arteritis (GCA). Their observation is concordant with our results, especially regarding the more frequent ophthalmic symptoms, lower biological inflammatory measures, and higher hemoglobin levels in patients with stroke2. Additionally, they observed more frequent large-vessel involvement in patients with stroke. Interestingly, they hypothesized the existence of a different pattern of the disease in these patients with more localized inflammation to the cephalic/supraaortic arterial tree and questioned the potential role of varicella zoster virus (VZV) as an etiological factor for GCA, given its known neurovascular tropism3,4. Three interesting points must … Address correspondence to Dr. H. de Boysson, Department of Internal Medicine, Caen University Hospital, University of Caen-Normandie, Avenue de la Cote de Nacre, 14000 Caen, France. E-mail: deboysson-h{at}chu-caen.fr
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