Abstract
In Parkinson's disease (PD), beta (β) and gamma (γ) oscillations are altered in the basal ganglia, and this abnormality contributes to bradykinesia pathophysiology. However, it is unclear whether β and γ rhythms at the primary motor cortex (M1) level influence bradykinesia. We aimed at clarifying this issue by using transcranial alternating current stimulation (tACS), a neurophysiological technique that enhances cortical rhythms by entraining endogenous oscillations.
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