Abstract

Acute retinal necrosis (ARN) occurs due to secondary reactivation of previous herpes simplex or varicella zoster infection. In children, ARN is rare and challenging to manage due to late presentation caused by difficulty in the communication of symptoms to caregivers and lack of treatment guidelines.[1] Acyclovir used for treatment can cause direct tubular injury or crystal deposit in the tubules, causing acute kidney injury (AKI). Prompt management of AKI is required to avoid lethal complications. In this case report, we describe the case of a 14-year-old boy with ARN secondary to primary varicella infection developing AKI following intravenous acyclovir.

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