Abstract
Objective: Apoptosis is essential to normal development as well as physiological cell turnover. Although apoptosis in excess can manifest as tissue damage, a failure to undergo apoptosis constitutes pathological cellular overgrowth. Nitric oxide (NO), a free radical with several physiologic functions, exhibits contradictory effects in the regulation of apoptosis and both pro- and anti-apoptotic effects have been demonstrated. This study examined the effect of different amounts of NO on apoptosis in mouse pre-implantation embryos.
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