Abstract
Introduction: Sickle-cell disease (SCD) is characterised by episodes of acute self-limiting pain due to vaso-occlusion. Around two-thirds of SCD patients are HbSS homozygotes whilst most of the remainder have HbSC disease. The HbSC phenotype is typically milder and patients experience fewer pain episodes. Whilst oral and maxillofacial pain and altered facial sensation have been reported in SCD, no genotype-specific data has been published.
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