Abstract
increase of A2A mRNA expression levels ( CTA2A 16,8±2.72 vs. 11,2±2.64; p<0.001), and a steady maintenance of the A1 mRNA levels. No differences between right and left atrium were observed. Conclusions: Sustained atrial high-rate stimulation induces an early overexpression of A2A receptors, suggesting an impairment of the cross-talk between atrial A1 and A2A receptors. This can be associated with a role for A2A receptors in atrial Ca2+ kinetics and atrial remodeling.
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