Abstract
CaV3.1 and CaV3.2 calcium channels serve critical roles in the cardiovascular and nervous systems. CaV3 dysregulation underlies diverse diseases, including chronic pain. Interventions that tune the functional expression and gating of CaV3 channels represent powerful mechanisms to regulate physiology, and opportunities for developing new therapeutics. Ubiquitination is a prevalent posttranslational modification that regulates different aspects of protein lifecycles, including subcellular localization and stability.
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