Abstract
Description of the topic: Erectile dysfunction is a prevalent condition among the male population and can result from various factors such as hormonal, metabolic, infectious, or anatomical issues. Infections caused by viruses such as Human Immunodeficiency Virus (HIV), Herpes Simplex Virus, Varicella-Zoster Virus, Human Papillomavirus (HPV), Human T-lymphotropic Virus type 1 (HTLV-1), Dengue virus, and SARS-CoV-2 have been found to be associated with erectile dysfunction through different physiological mechanisms. In many cases, these infections interact with the immune system, leading to the development of a localized or generalized inflammatory response of varying intensity. Relevance: Certain viral infections can directly affect the tissues of the male reproductive system, resulting in erectile dysfunction. These infections can damage the nerves and blood vessels that are essential for achieving and maintaining an erection. Conclusion: Erectile dysfunction is of multifactorial origin, and according to the reviewed literature it can be triggered by some vascular sequelae induced by viral infections, including those produced by the acquired immunodeficiency virus (HIV), herpes simplex virus, zoster, human papilloma (HPV), human lymphotropic virus type I (HTL1), Dengue and SARS-CoV2.
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