Abstract
In the not too distant past, the research consensus was that attention-deficit/hyperactivity disorder (ADHD) is a pathophysiologically homogeneous condition: localize the site of the core brain deficit shared in common by ADHD patients, we thought, and we will have solved the riddle of the disorder. A number of hypotheses regarding what the core to ADHD might be were proposed. Of these, the notion that ADHD is a disorder of higher order executive control has carried particular weight. Especially influential has been the idea, prefigured in other theories but most effectively crystallized by Barkley (1), that executive dysfunction in ADHD is mediated by alterations in frontostriatal inhibitory processes.
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