Abstract
Unilobar injection of starch solution produced pulmonary infarction at the site of injection and diffuse bilateral pulmonary edema. The predominantly or entirely unilobar distribution of the embolizing material was established by histologic methods and by a technic using whole lung lobe suspension stained with Lugol's solution. Evidence is presented suggesting that the pulmonary edema in the nonembolized lobes is attributable to neural or neurohumoral mechanisms.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
