Abstract
T helper cells are thought to be of importance in the pathogenesis of atopic dermatitis (AD)(1,2). Most patients with (AD) have elevated serum IgE levels(2–4). Allergens stimulate a certain population of T helper cells, the Th2-cells, that produce high amounts of interleukin-4 (IL-4), and relative low amounts of interferon-γ (IFN-γ)(5). IL-4 stimulates B-cells to IgE production(6). Bacteria and viruses induce the activation of Thl-cells, resulting in IFN-γ production and as a result we see a delayed hypersensitivity reaction (e.g.)(7) KeywordsAtopic DermatiHigh Gene ExpressionActa DermDelay Hypersensitivity ReactionAtopic Dermati SkinThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
