Abstract
Conclusions: We have established cultures for three cell populations involved in flexor tendon wound healing: synovial sheath fibroblasts, epitenon tenocytes, and endotenon tenocytes. The active gene expression of TGF-Beta isoforms by both extrinsic synovial sheath fibroblasts and intrinsic tenocytes [epitenon and endotenon] suggests dual mechanisms of flexor tendon wound healing which may be modulated to promote scarless tendon healing.
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