Abstract
In B-type neurones of bullfrog sympathetic ganglia, apamin (10 nM) suppressed the Ca 2+-dependent K + current (I AH) involved in the afterhyperpolarization of an action potential, while it did not affect the Ca 2+-dependent K + current (I C) underlying the spike repolarization. I AH was further separated into two exponential components which were differentially affected by apamin, voltage and alterations in Ca 2+ influx, suggesting the existence of 3 different types of Ca 2+-dependent K + channel in bullfrog sympathetic neurones.
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