Abstract
The assumption that the α-chain gene is duplicated and that there are potentially two genes for α-thalassæmia per chromosome, one for each α-chain gene, can explain, among other things, why in some populations a high incidence of hæmoglobin Bart's (γ 4) can occur in newborn children without the correspondingly expected appearance of hæmoglobin H disease and the hæmoglobin Bart's hydrops fetalis syndrome.
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