Diets, food and idiopathic Parkinson`s disease

José Perea Sasiaín,Roberta Hanfling Schwartz

doi:10.15446/revfacmed.v62n1.43783

José Perea Sasiaín, Roberta Hanfling Schwartz

Open Access

https://doi.org/10.15446/revfacmed.v62n1.43783

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Abstract

Based on two individual cases, this is a personal depict on knowledge of Idiopathic Parkinson´s Disease (IPD) etiopathogeny emphasizing diets, food and edible substances tested and reported. In the first case (adherence to an almost vegan diet, with one seventh of the protein supplied from animal origin), had an impressive beneficial effect on his fifteen years clinical course, while in the second case (continuation of a quasi ovo-lacto-vegetarian diet), did not modify substantially the clinical course. The relevance of diet on IPD focused in redistribution of protein daily intake when levodopa prescribed. Diet by itself is fundamental for the management of each individual person prone to or suffering from IPD. Vegan diets with pulses rich in protein duly balanced with cereals and supplemented with vitamin B12, reduce the intake of methionine and keep the amount of aromatic amino acids at a moderate level.

Highlights

The six patients described by James Parkinson [1] as afflicted by Shaking Palsy, motivated Jean Marie Charcot to label them as suffering Parkinsons disease [2], yet this syndrome would have been a better choice with its classic triad: tremor, stiffness and slowness
Palacios [12] reported that Idiopathic Parkinsons Disease (IPD) patients were heavier and had more fat in their bodies: this can be assume as the result of overeating and/or not enough physical exercise
But in-depth, personal experiences, diet has a primary importance and must be the first step in prevention and treatment of IPD: it is fundamental for people (1-2%) enduring, or susceptible to, IPD invalidating conditions

Summary

Introduction

The six patients described by James Parkinson [1] as afflicted by Shaking Palsy, motivated Jean Marie Charcot to label them as suffering Parkinsons disease [2], yet this syndrome would have been a better choice with its classic triad: tremor, stiffness and slowness. A large number of other non-motor afflictions add to the protracted clinical course of each patient. A partir de dos casos individuales, se presenta un esbozo personal sobre el conocimiento de la etiopatogenia en la Enfermedad de Parkinson Idiopática (EPI), con un énfasis en las dietas, la comida y las sustancias comestibles probadas y reportadas. La relevancia de Idiopathic Parkinsons Disease (IPD) is more prevalent in countries with high industrial development and high economic standards. Nowadays it is apparent that there are several distinct types of parkinsonian disorders [3] and each patient individually endures his lot

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