Abstract
Neuropathy is a common long-term complication of diabetes recognized almost entirely in adults (Moser et al., 2013). It is a precursor to foot ulcers and lower limb amputations, both of which are preventable (Fletcher, 2006; Zangaro & Margaret, 1999). The pathogenesis of diabetic peripheral neuropathy (DPN) is not completely understood. Evidence suggests that hyperglycemia and other metabolic factors contribute to nerve damage, at least in part by degrading the function of small blood vessels supplying the nerves, resulting in progressive structural deterioration of nerve pathways (Trotta, Verrotti, Salladini, & Chiarelli, 2004).
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