Abstract
INTRODUCTION Previous studies have shown a decreased body weight and a greatly reduced pancreatic insulin content in the offspring of manifest diabetic rat mothers (1). These findings differ from those in human diabetic fetopathia which is characterized by an increased body weight, elevated levels of circulating insu- lin and an increased fetal pancreatic insulin reserve (2). It is nevertheless clear, that manifest diabetes in the pregnant rat gives rise to a variety of disturbances in fetal development. These alterations include not only those changes mentioned above, but also an arrest, rather than a stimulation of the pancreatic B-cell growth
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