Abstract
Selective IgA deficiency has been associated with the use of various therapeutic agents such as phenytoin, D-penicillamine, and gold. ~'2 In some cases, immunoglobulin A concentrations returned B normal after the drug was discontinued. Recently, sulfasalazine therapy has been associated with selective IgA deficiency in adults given treatment for rheumatoid arthritis 3 and in children given treatment for inflammatory bowel diseases?,5 Some, but not all, patients demonstrated increased IgA levels after discontinuation of sulfasalazine. We report a case in which the use of sulfasalazin e for the therapy of ulcerative colitis was followed by the development not only of persistent IgA deficiency, but of IgG2 subclass deficiency as well.
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