Abstract
The purpose of this study was to determine the effects of ten weeks of high intensity interval training (HIIT) with ginger supplementation on the systemic inflammatory response to acute HIIT exercise.Twenty four sedentary overweight women randomly assigned into three groups’ including HIIT + ginger (n=8), HIIT + placebo (n=8) and ginger (n=8). Sixteen women performed ten weeks of HIIT while the supplement consuming groups daily took 3 gm. of ginger pills and the HIIT + placebo group took placebo. Serum samples were collected at rest and immediately post-acute exercise, before and after the training. Two-way repeated measures ANOVA, paired t test, one-way ANOVA and post hoc benferoni were used to analyze data. Results indicated that three groups similarly affect the Monocyte Chemo tactic proetein-1 (MCP-1), Interlukine-10 (IL-10) changes in response to acute exercise (p>0.05) but showed significant increase in intercellular adhesion molecule-1 (ICAM-1) in HIIT + placebo group in comparison with HIIT + ginger group (p=0.02) in second acute exercise. The combination of HIIT and ginger consumption may induce a modest inflammatory attenuation of atherosclerosis factors in response to acute exercise and improvement in Vo2max and PBF results rather than ginger and training alone, in young sedentary overweight women.
Highlights
Scientific documents agree the hypothesis that functions of inflammation in the initiation, progression and plaque rupture of the acute coronary disease, atherosclerosis (Libby et al, 2002)
On the other hand, paired-t test confirmed significant increase of maximum oxygen consumption (Vo2max) in high intensity interval training (HIIT) + ginger and HIIT + placebo compared to pre-test
We sought to examine the effect of 10 weeks of short-term high-intensity interval training (40-m maximal Shuttle run) induced changes in serum intercellular adhesion molecule-1 (ICAM-1), Monocyte Chemo tactic protein-1 (MCP-1) and IL-10 levels following an acute highintensity interval exercise challenge performed at the same absolute intensities as those undertaken prior to training
Summary
Scientific documents agree the hypothesis that functions of inflammation in the initiation, progression and plaque rupture of the acute coronary disease, atherosclerosis (Libby et al, 2002). In this phenomena, a variety of inflammatory biomarkers have been identified, some of them act as primary markers in the first stages of progression. (MCP-1) an inflammatory chemokine, respectively (Ribeiro et al, 2012). It seems that some of athero-protective agents are present in the lesion to attenuate the inflammatory cascade. Available evidence suggests that concentration of ICAM1, MCP-1and IL-10 can be modulating by pro inflammatory conditions (Vrousgos, 2015), exercise training (Ribeiro et al, 2012) or after coupling with some of natural ingredients of herbs as seen by ginger (Wang et al, 2013)
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