Dentin hypersensitivity after treatment with desensitizing agents: a randomized, double-blind, split-mouth clinical trial

Jorgiana Silva De Assis,Sérgio Lima Santiago,Cristiane Sá Roriz Fonteles,Lidiany Karla Azevedo Rodrigues,Regina Cláudia Ramos Colares,André Mattos Brito De Souza

doi:10.1590/s0103-64402011000200012

Jorgiana Silva De Assis, Sérgio Lima Santiago + Show 4 more

Open Access

https://doi.org/10.1590/s0103-64402011000200012

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Abstract

The aim of this study was to evaluate the efficacy of two desensitizing agents in the reduction of dentin hypersensitivity in a randomized, double-blind, split-mouth clinical trial. Seventy-seven teeth from 13 patients that presented some degree of sensitivity to probing and/or air stimulation were treated with one of the following desensitizing agents: Oxa-Gel (G1), Sensi Kill (G2) and placebo gel (G3 - control). According to paired t-test, all treatments, even the placebo gel, were capable of reducing sensitivity scores for both stimuli. Analysis of data by ANOVA and Tukey's test (?=0.05) showed that the sensitivity scores were significantly lower only for Sensi Kill in comparison to the other products (Oxa-Gel and placebo), when air stimulus was applied. It may be concluded that treatment with Sensi Kill presented a slightly better performance in reducing dentin hypersensitivity when compared to the other desensitizing agent.

Highlights

Dentin hypersensitivity can be defined as a temporary pain or exaggerated response from the exposed dentin to chemical, tactile, thermal or osmotic stimuli in the buccal environment, which would not normally occur in a healthy tooth [1]
The aim of this study was to evaluate the efficacy of two desensitizing agents in the reduction of dentin hypersensitivity in a randomized, double-blind, splitmouth clinical trial, to test the hypothesis that one of desensitizing agent is better than the other
According to ANOVA, sensitivity scores were significantly lower (p

Summary

Introduction

Dentin hypersensitivity can be defined as a temporary pain or exaggerated response from the exposed dentin to chemical, tactile, thermal or osmotic stimuli in the buccal environment, which would not normally occur in a healthy tooth [1]. The exposure of dentin tubules due to enamel loss by abrasion, erosion, abfraction or root surface exposure caused by gingival recession, periodontal treatment or a combination of both [2] may produce strong dentin sensitivity [3]. It has been reported that 8 to 30% of adults are affected by dentin hypersensitivity [8,9]. Females have been reported to have a higher incidence of hypersensitivity than males, the difference is not statistically significant, and the greatest incidence has been documented in the 20 to 40 year-old group [8].

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