Abstract
Abstract: A 25-year-old man was referred for a bone scan to assess sacral pain on a background of β-thalassemia major complicated by severe anemia. He required repeated blood transfusions resulting in iron overload, which was treated with long-term iron chelation therapy. This case illustrates the potential skeletal manifestations of thalassemia and its treatment as demonstrated on bone scintigraphy, which include alterations in skeletal development and maturation, particularly delayed epiphyseal closure as well as complications of severe osteoporosis. Although the causes are multifactorial, pituitary iron deposition complicated by hypogonadotrophic hypogonadism should be considered as these patients may benefit from hormone replacement.
Published Version
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