Abstract
Background: Meconium aspiration syndrome (MAS) is a significant problem in term and near-term neonates. Meconium has been shown to induce apoptotic death of alveolar epithelial cells. It is known that meconium contains pancreatic and intestine brush border enzymes which might be responsible for acute lung damage observed in MAS. Previous studies from our lab showed that ACE-2 is downregulated in experimental lung fibrosis. The main function of ACE-2 is to convert the octapeptide ANG II to the heptapeptide ANG 1-7. ANG II is a …
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