Abstract
The defective interfering (DI) RNAs represent one of several classes of symptom-modulating RNAs identified in association with RNA plant virus infections. Structurally, these molecules are derived from, and represent mutant forms of, the viral genome (Perrault 1981; Lazzarini et al. 1981). DI RNAs may contain distinct types of modifications; however, the most prevalent is the deletion of one or more large segments of sequence. Despite structural differences, the common effect of the mutation(s) is to render the DI RNAs dependent on their nondefective ‘parental’ genome for essential viral replication proteins. This, in turn, limits replication of these molecules to cells which are coinfected with the parental genome.KeywordsCitrus Tristeza VirusRingspot VirusHelper VirusJunction SiteTomato Bushy Stunt VirusThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.
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