Abstract
The complement system is central to innate immunity and has evolved primarily for microbial destruction. Yet, its non-specific nature requires tight regulation to prevent host cell complement-induced damage. Complement regulatory proteins are proposed to execute this function. Moreover, such proteins are essential for pregnancy maintenance in mice (Science 2000;287:498). This study was conducted to determine whether preterm labor with histologic chorioamnionitis is associated with changes in the expression of complement regulatory proteins CD46 (membrane cofactor protein/MCP) and CD59 (protectin) in human chorioamniotic membranes.
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