Abstract

Posterior reversible encephalopathy syndrome (PRES) is a neurological disorder characterized by its reversibility both radiologically and clinically, and it typically has a favorable prognosis. Its etiopathogenesis is not clear, although it is known that it involves endothelial dysfunction of the posterior cerebral vasculature, leading to failed cerebral autoregulation. We report a patient with PRES that may have been caused by an immune-mediated response to decitabine. Key words: Posterior reversible encephalopathy; Decitabine.

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