Abstract
David M. Livingston left us on October 17, 2021. He was best known for his fundamental work on retinoblastoma protein and its effects on cell-cycle progression and, in recent decades, on the biochemistry and function of the homologous repair-associated BRCA1 and BRCA2 proteins. The latter, as we now know, contribute to the pathogenesis of a significant fraction of human breast cancers. Their roles in tumor pathogenesis are derived in large part from their function as mediators of the DNA repair process of homologous recombination.
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