Abstract
TEMPI syndrome is an acquired plasma cell dyscrasia characterised by telangiectasias of the face, elevated erythropoietin and erythrocytosis, monoclonal gammopathy, perinephric fluid collections and intrapulmonary shunting (1). The first case of TEMPI syndrome was described by Bazari at al. in 2010 (2) and currently 23 cases have been identified worldwide (3, 4).
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