Abstract
Aims: The pathophysiology of migraine is not yet fully understood and stroke has to be included in differential diagnosis. The neurogenic theory is confirmed by neuroimaging studies with functional MRI during auras indicating that the decreases in cortical bloodflow are not sufficient to cause ischemia and the subsequent vasodilatation is observed well after the beginning of headache. Neuroimaging descriptions during migraine attacks are limited to MRI, but this technology is not widely available on a 24h basis. Recent developments in CT technology allows high speed volumetric data acquisition and generation of perfusion maps.
Published Version
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