Abstract
[1] Takemura G, Nakagawa M, Kanamori H, et al. Benefits of reperfusion beyond infarction size limitation. Cardiovasc Res 2009;83:269–76. [2] Frangoginis NG, Smith CW, Entman ML. The inflammatory response in myocardial infarction. Cardiovasc Res 2002;53:31–47. [3] Scaffidi P, Misteli T, Bianchi ME. Release of chromatin protein HMGB1 by necrotic cells triggers inflammation. Nature 2002;418:191–5. [4] Bell CW, Jiang W, Reich CF, et al. The extracellular release of HMGB1 during apoptotic cell death. Am J Physiol Cell Physiol 2006;291:C1318–25. [5] Andrassy M, Volz HC, Igwe JC, et al. High-mobility group box-1 in ischemia– reperfusion injury of the heart. Circulation 2008;117:3216–26. [6] Jiang WL, Zhang SP, Zhu HB, et al. Cardioprotection of asperosaponin X on experimental myocardial ischemia injury. Int J Cardiol 2012;155:430–6. [7] Hu X, Zhou X, Xu C, et al. Minocycline protects against myocardial ischemia and reperfusion injury by inhibiting high mobility group box 1 protein in rats. Eur J Pharmacol 2011;654:274–9. [8] Hu X, Cui B, Zhou X, et al. Ethyl pyruvate reduces myocardial ischemia and reperfusion injury by inhibiting high mobility group box 1 protein in rats. Mol Biol Rep 2012;19:227–31. [9] Hu X, Fu W, Jiang H. HMGB1: a potential therapeutic target for myocardial ischemia and reperfusion injury. Int J Cardiol 2012;155:489-489. [10] Abraham NG, Kappas A. Heme oxygenase and the cardiovascular-renal system. Free Radic Biol Med 2005;39:1–25. [11] Ryter SW, Alam J, Choi AM. Heme oxygenase-1/carbon monoxide: from basic science to therapeutic applications. Physiol Rev 2006;86:583–650. [12] Liu SX, Zhang Y, Wang YF, et al. Upregulating of heme oxyenase-1 expression by hydroxysafflor yellow A conferring protection from anoxia/reoxygenation-induced apoptosis in H9c2 cardiomyocytes. Int J Cardiol 2012;160:95–101. [13] Takamiya R, Hung CC, Hall SR, et al. High-mobility group box 1 contributes to lethality of endotoxemia in heme oxygenase-1-deficient mice. Am J Respir Cell Mol Biol 2009;41:129–35. [14] Tsoyi K, Lee TY, Lee YS, et al. Heme-oxygenase-1 induction and carbon monoxidereleasingmolecule inhibit lipopolysaccharide (LPS)-induced high-mobility group box 1 release in vitro and improve survival of mice in LPSand cecal ligation and punctureinduced sepsis model in vivo. Mol Pharmacol 2009;76:173–82.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.